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Year Number of Results
1987 7
1988 25
1989 31
1990 61
1991 67
1992 152
1993 242
1994 280
1995 285
1996 331
1997 348
1998 325
1999 365
2000 371
2001 411
2002 397
2003 441
2004 427
2005 436
2006 479
2007 500
2008 486
2009 414
2010 446
2011 466
2012 429
2013 423
2014 415
2015 360
2016 323
2017 307
2018 254
2019 259
2020 297
2021 292
2022 259
2023 223
2024 64

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10,697 results

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The following term was not found in PubMed: 242816
Page 1
The PLAUR signaling promotes chronic pruritus.
Chen W, Li Y, Steinhoff M, Zhang W, Buddenkotte J, Buhl T, Zhu R, Yan X, Lu Z, Xiao S, Wang J, Meng J. Chen W, et al. FASEB J. 2022 Jun;36(6):e22368. doi: 10.1096/fj.202200079R. FASEB J. 2022. PMID: 35596683 Free PMC article.
In this study, the clinical relevance of a putative Serpin E1 receptor PLAUR to chronic itch, and the neuro-cutaneous Serpin E1-PLAUR signaling are explored. ...OSM induced acute itch in mice and promoted G-CSF and IL-8 release from human kerati …
In this study, the clinical relevance of a putative Serpin E1 receptor PLAUR to chronic itch, and the neuro-cutaneous Serpi …
Serpin E1 mediates the induction of renal tubular degeneration and premature senescence upon diabetic insult.
Chen BH, Lu XQ, Liang XH, Wang P. Chen BH, et al. Sci Rep. 2023 Sep 27;13(1):16210. doi: 10.1038/s41598-023-43411-4. Sci Rep. 2023. PMID: 37758806 Free PMC article.
Reminiscent of the action of typical senolytics, a small molecule inhibitor of Serpin E1 substantially mitigated the pro-senescent and degenerating effects of the diabetic milieu, suggesting an essential role of Serpin E1 in mediating renal tubular sen …
Reminiscent of the action of typical senolytics, a small molecule inhibitor of Serpin E1 substantially mitigated the pro-senes …
Forestalling fibrosis.
Ingelfinger JR. Ingelfinger JR. N Engl J Med. 2003 Dec 4;349(23):2265-6. doi: 10.1056/NEJMcibr032288. N Engl J Med. 2003. PMID: 14657436 Review. No abstract available.
Interplay of Helicobacter pylori, fibroblasts, and cancer cells induces fibroblast activation and serpin E1 expression by cancer cells to promote gastric tumorigenesis.
Chen X, Chen W, Zhao Y, Wang Q, Wang W, Xiang Y, Yuan H, Xie Y, Zhou J. Chen X, et al. J Transl Med. 2022 Jul 21;20(1):322. doi: 10.1186/s12967-022-03537-x. J Transl Med. 2022. PMID: 35864535 Free PMC article.
Finally, Serpin E1 and/or FAP expression was measured in H. pylori-infected gerbil gastric mucosa and human gastric cancer tissues. ...The interplay of H. pylori, fibroblasts, and cancer cells promotes the transition of NFs to CAFs by inducing cytokine releas …
Finally, Serpin E1 and/or FAP expression was measured in H. pylori-infected gerbil gastric mucosa and human gastric can …
Plasminogen activator inhibitor-1.
Gils A, Declerck PJ. Gils A, et al. Curr Med Chem. 2004 Sep;11(17):2323-34. doi: 10.2174/0929867043364595. Curr Med Chem. 2004. PMID: 15379715 Review.
Plasminogen activator inhibitor-1 (PAI-1) is an important component of the plasminogen/plasmin system as it is the main inhibitor of tissue-type and urokinase-type plasminogen activator. Consequently, PAI-1 plays an important role in cardiovascular dis …
Plasminogen activator inhibitor-1 (PAI-1) is an important component of the plasminogen/plasmin system as it is t …
Growth differentiation factor myostatin regulates epithelial-mesenchymal transition genes and enhances invasion by increasing serine protease inhibitors E1 and E2 in human trophoblast cells.
AbdelHafez FF, Klausen C, Zhu H, Yi Y, Leung PCK. AbdelHafez FF, et al. FASEB J. 2023 Oct;37(10):e23204. doi: 10.1096/fj.202300740R. FASEB J. 2023. PMID: 37738042
Considering that the mechanistic role of MSTN in placentation remains poorly understood, we hypothesized that MSTN uses ALK4/5-SMAD2/3/4 signaling to increase human trophoblast invasion through a group of epithelial-mesenchymal transition genes including SERPINE2, PAI-1, a …
Considering that the mechanistic role of MSTN in placentation remains poorly understood, we hypothesized that MSTN uses ALK4/5-SMAD2/3/4 sig …
PAI-1 and atherothrombosis.
Vaughan DE. Vaughan DE. J Thromb Haemost. 2005 Aug;3(8):1879-83. doi: 10.1111/j.1538-7836.2005.01420.x. J Thromb Haemost. 2005. PMID: 16102055 Free article. Review.
Plasminogen activator inhibitor-1 (PAI-1) is the major physiologic inhibitor of tissue-type plasminogen activator in plasma, and is elevated in a variety of clinical situations that are associated with increased risk of ischemic cardiovascular events. …
Plasminogen activator inhibitor-1 (PAI-1) is the major physiologic inhibitor of tissue-type plasminogen activato …
RBM4 regulates cellular senescence via miR1244/SERPINE1 axis.
Wang L, Zhang X, Sheng J, Chen L, Zhi L, Zheng Q, Qi Y, Wang L, Zhang J, Zhao J, Wang Y, Liu SX, Sun MZ, Zhang W. Wang L, et al. Cell Death Dis. 2023 Jan 13;14(1):27. doi: 10.1038/s41419-023-05563-z. Cell Death Dis. 2023. PMID: 36639375 Free PMC article.
Plasminogen activator inhibitor-1 in cancer research.
Li S, Wei X, He J, Tian X, Yuan S, Sun L. Li S, et al. Biomed Pharmacother. 2018 Sep;105:83-94. doi: 10.1016/j.biopha.2018.05.119. Epub 2018 May 28. Biomed Pharmacother. 2018. PMID: 29852393 Review.
[Despite as a major inhibitor of urokinase (uPA), paradoxically,] Plasminogen activator inhibitor-1 (PAI-1) has been validated to be highly expressed in various types of tumor biopsy tissues or plasma compared with controls based on huge clinical data …
[Despite as a major inhibitor of urokinase (uPA), paradoxically,] Plasminogen activator inhibitor-1 (PAI-1) has …
A slice of PAI.
Loskutoff DJ. Loskutoff DJ. J Clin Invest. 1993 Dec;92(6):2563. doi: 10.1172/JCI116866. J Clin Invest. 1993. PMID: 8254010 Free PMC article. Review. No abstract available.
10,697 results
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